Genes that trigger the development of neurological diseases - Database & Sql Blog Articles

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A new study from Yale University and others pointed out that mutations in a gene that controls lipid metabolism may trigger a slow-progressive neurogenic disease characterized by myelin, a related study or to reveal multiple control The new treatment for sexual sclerosis provides ideas, and the study was published in the international journal Nature Cell Biology .

In the article, the researchers point out that a single gene mutation called FAM126A triggers a series of pathological lesions, such as developmental delay, mental retardation, peripheral neuropathy, and muscle atrophy. It also causes congenital cataracts, but Researchers are not aware of the precise function of the gene.

Researcher Pietro De Camilli said that we found that the protein hyccin encoded by the FAM126A gene can help produce a lipid that is important for myelination, which surrounds and protects axons in neuronal cells throughout the nervous system; In this study, researchers combined with researchers from the United States, Italy, and other countries to analyze cells in patients with myelin degeneration and congenital cataract found that mutations in the gene FAM126A lead to enzyme complexes that are important for myelin production. Loss of stability.

In patients with multiple sclerosis, the progression of the disease mainly depends on the re-generation of myelin, and the immune system of the patient's body often attacks the myelin sheath, eventually leading to neuronal death; the researchers speculate that the body is developing normally. Hyccin plays a crucial role in the production of myelin in the recovery phase of patients with multiple sclerosis; in the later studies, researchers may conduct extensive research on a large number of samples to develop neurological diseases such as multiple sclerosis. The new therapies provide a basis for research.

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